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AST Bravo II 3/25s & 4/33 Driver
It is a kind of compliment which they have a right to, and I am always glad when I can pay it. Sometimes, however, it is no easy thing.
Many a page have I turned over and over without being able to find anything to my purpose; but I never yet AST Bravo II 3/25s & 4/33 over three in Shakspeare without hitting upon just what I wanted. Putnam,p. Title page epigraphs are added from a number of other works by both authors.
I should have preferred to transcribe the epigraphs as well as giving their sources, but this would take too much space. Notice is taken of misattributions, and significant alterations in quoted texts.
Susan Fenimore Cooper 1. Carey and Hart,by chapter and author.
Title page epigraph from Rural Hours New York: George P. Putnam, Texts of epigraphs as yet unidentified; please help us find them! Citations identify: Act, Book, or Canto large Roman numerals ; scene or verse small roman numerals ; line numbers Arabic numerals.
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Where Cooper misattributed an epigraph, or his text differs markedly from the standard version, his attribution or words are shown following the Part II citation. A few errors have been corrected. Henry Holt, Wayne Franklin of Northeastern University, and Ms.
Michaelyn Burnette of the Library at the University of California at Berkeley, have both assisted in tracking down epigraphs from sources not available in Cooperstown. My thanks to all. The editions from which epigraphs were taken can only rarely be identified.
The Cooper Epigraphs
James Fenimore Cooper's eight-volume "travelling set" of Shakespeare London: Nichols et al. Some would contend that improvements in AML occur in small increments and basing stopping treatment on larger increments is unrealistic and counterproductive. Others would counter that the number of patients and amount time of AST Bravo II 3/25s & 4/33 needed to detect small improvements is far from desirable. This issue is likely to demand further discussion as the recognition that AML is several diseases each to be treated differently inevitably will mean fewer patients available for the various trials.
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It would seem that at least logistically the specified degree of improvement will have to be higher than currently, perhaps justified by the increased specificity of new therapies. These issues aside it seems quite likely that our results in newly-diagnosed patients are not comparably satisfactory as those of Advani et al.
A possible biologic explanation is that high intracellular cholesterol may figure more prominently as an adaptive mechanism leading to chemoresistance in relapsed AML and therefore the value of statin addition may be less in newly-diagnosed patients. Obviously, it is also possible that patient and disease characteristics may affect the outcome in AML more than treatment, here IAP.